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Lipopolysaccharide (LPS), a prototypical bacterial endotoxin, has been identified as a key mediator linking microbial dysbiosis to GLP-1 activation

Wang XW, Zhang H, Sapio R, et al

In our last post, we covered the lifestyle decisions that can enhance our glutathione

insulin is one of the most well-known examples, and modern metabolic medications like GLP-1 receptor agonists have transformed how we treat diabetes and obesity

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Both outcomes are clinically meaningful, but they illustrate why BMI at treatment initiation shapes expectations about results