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Hence, the binding of LDL to proteoglycans makes the LDL more susceptible to oxidation and aggregation, which promotes foam cell formation and a proinflammatory response, and the process is self-perpetuating

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This recruitment is orchestrated by chemokines, adhesion molecules, and other signaling molecules released by the inflamed tissue (77).Understanding the complex interplay between immune cell recruitment and oxidative stress in IBD is crucial for developing targeted therapies