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Furthermore, a decrease in total GST activity advanced with MASLD progression in humans (Hardwick et al

The extrinsic apoptosis pathway is triggered by death receptor-death ligand interactions, including FasL/FasR, TRAIL/TRAIL-R1 or TRAIL-R2, and TNF/TNF-R1 [77,78,79]

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It is a GLP-1 receptor agonist and works by stimulating insulin secretion, suppressing glucagon secretion, reducing appetite, and slowing gastric emptying

It is commonly used in studies focused on endocrine signaling, receptor interaction, and hormone pathway modeling

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