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Deubiquitination of active NLRP3 triggers ASC aggregation and the recruitment of precaspase-1, which leads to the formation of the NLRP3 inflammasome and the release of proinflammatory cytokines such as IL-1 and IL-18

In contrast, during chronic or infected wound states, the pH shifts to alkaline levels (pH 79) as a result of microbial activity 53,54

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