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Myocardial infarction models have documented reduced infarct sizes and improved cardiac function when GHK-Cu is administered shortly following coronary occlusion, effects attributed to enhanced angiogenesis, reduced inflammatory damage, and improved matrix remodeling in healing myocardium

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The copper(II) complex can hydrolyze slowly under alkaline conditions, releasing free copper ions that do not carry the peptide's signaling activity and can cause localized irritation

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