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The increase of this pro-inflammatory state, in turn, also maintains OS and disables the EAS in a loop process that will remain active indefinitely, inducing apoptosis of melanocytes without its proper replacement leading to vitiligo scars and disease progression through time (Ortona et al., 2014

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Additionally, we observed an induction of iron regulatory protein 2 (IRP2), a major component of the iron response pathway which is activated upon iron-sulfur cluster synthesis dysfunction 30 (Fig

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