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We further show that overexpression of enzymes or supplementation of key metabolites in the transsulfuration pathway of SAA metabolism significantly restores the sensitivity of HNF4-depleted liver cancer cells to methionine/cystine restriction or sorafenib treatment and inhibits cell migration (Fig

e , SMART1 inhibited Smurf1 protein level and PDK1 neddylation in cells (Smurf1-L: 0.5 M, SMART1Et: 0.5 M or SMART1: 0.5 M)

The GPx activity was calibrated using a reference method (Rotruck et al

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