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However, the level of systemic gut microbiota-dependent TMAO has been linked to mortality, regardless of the cause of permeability (hypoxemia or pro-inflammatory state) (179)

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To assess if cells with an undifferentiated or MES-like phenotype were affected, we performed immunostaining for the embryonic marker SOX9 (SRY-box transcription factor 9) and the marker of adrenergic (ADR) neuron differentiation TH (tyrosine hydrolase) 12

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