In reverse mode NET causes an efflux of the cytoplasmic pool of presynaptic norepinephrine into the synaptic cleft, where it can activate postsynaptic -1 and -1 receptors to elevate systemic vascular resistance and stimulate cardiac output (Vansal & Feller 1999
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1 ), clinically ( Figs
Preliminary clinical research found that individuals with coronary endothelial dysfunction (an early sign of atherosclerosis where blood vessels dont dilate properly) had significantly lower circulating MOTS-c levels compared to healthy individuals
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