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Regulation of AMP-activated protein kinase by multisite phosphorylation in response to agents that elevate cellular cAMP

In addition, we show that, the liganded -CT forms a rigid beam-like structure with the unliganded -CT, which prevents its rearrangement required for the binding of a second IGF1 molecule

However, GLP-1, in addition to various GLP-1R agonists, is a 300-fold stronger inducer of GSIS (4, 9) and does not produce the effects of glucagon to increase hepatic glucose production, which is thought to be a key pathogenic driver of T2DM (3)

Journal of Biomaterials Science, Polymer Edition, 19(8), 969988