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Azithromycin attenuates myofibroblast differentiation and lung fibrosis development through proteasomal degradation of NOX4

The expulsed chromatin, coated with antimicrobial proteins, forms NETs (neutrophil extracellular traps), able not only to trap pathogenic microorganisms, but also to propagate inflammation and to favour the induction of microclots following platelet binding (144, 145)

(19) studied the possible association between MnSOD and GPX1 gene polymorphisms and susceptibility and disease progression of alopecia areata in a Turkish population

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