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Prominent lymphocytic inflammation, many genetic susceptibility variants involving immune pathways, as well as potent responses of the neuroinflammatory component to immunomodulating drugs, have led to the natural conclusion that this disease is driven by a primary autoimmune process

Rats were placed in plastic startle tubes and positioned on a weight transducing platform enclosed in a sound-attenuating chamber

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HA + GSH-C4 exposure was associated with transient elevations in selected mediators (e.g., IFN-2, IFN-, TNF-, IL-6, IL-23, IL-33), whereas HA-only produced minimal changes