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There is evidence for clonal expansion of T cells in the CSF of AD patients, indicating an antigen-driven response in the CNS

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This microenvironment-driven conversion is supported by the transcriptional upregulation of key stemness regulators such as OCT4, NANOG, and SOX2 and is further reinforced by epigenetic modifications such as promoter methylation or histone acetylation, which stabilize the reprogrammed state

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