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BPC-157 upregulates growth hormone receptors on fibroblasts (the cells that produce collagen)

Thus, the ultimate clinical phenotype in a disease where both white matter cytodegeneration and adaptive autoimmune inflammation conspire in the pathogenesis is determined by the vigor of either or both processes, i.e., by a convolution of the two

Available information derives from animal studies or from mechanistic reasoning

Growth Hormone & IGF Research (1999) 10

Include leptin-deficient cohorts if mechanistic generalizability is a goal