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However, N-terminal modifications, such as N-acetylation, D-norleucine substitution, or replacement with -aminobutyric acid (GABA), dramatically elongated the peptides half-lives

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Therefore, we used a model of spinal cord injury that has many characteristics found in human spastic syndrome [42] and can be used long-term to provide a realistic model of spasticity development in the tail muscle

The pathophysiology of migraine is complex, and the role of genetic, metabolic, and hormonal factors has been proposed (195)