Lipopolysaccharide (LPS) models, in which neuroinflammation is induced by LPS injection into the brain, are attractive given the central role of glia and neuroinflammation in AD 151
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In OA, this balance is disrupted by catabolic signaling, inflammation, and mechanical stress, culminating in progressive cartilage erosion and exposure of subchondral bone (Peng Z
[DOI] [PubMed] [Google Scholar] 47.Girardin M, Dionne S, Goyette P, Rioux J, Bitton A, Elimrani I, Charlebois P, Qureshi I, Levy E, Seidman EG
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