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The exact cause of neuronal death in Parkinson's remains incompletely understood, but contributing factors include oxidative stress, mitochondrial dysfunction, neuroinflammation, and abnormal protein aggregation (particularly alpha-synuclein)

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Antioxidant enzyme support: Research demonstrates the compound increases superoxide dismutase (SOD), catalase, and glutathione peroxidase activity, reducing oxidative DNA damage and lipid peroxidation markers in aged animal models

Studies show persistent elevation of vascular injury markers such as vWF and cytokines, suggesting ongoing thromboinflammation and hypercoagulable states in Long COVID [362, 363]

PPAR-3KO-CD, n = 6