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Nonanatomical contributors to OSA, such a low threshold for arousal, are likely to be particularly important in OSA pathogenesis in nonobese patients with OSA.

Liu L, Liu J, Wong WT, Tian XY, Lau CW, Wang YX, Xu G, Pu Y, Zhu Z, Xu A, et al: Dipeptidyl peptidase 4 inhibitor sitagliptin protects endothelial function in hypertension through a glucagon-like peptide 1-dependent mechanism

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Postmenopausal bone loss accelerates in the first 35 years after menopause precisely the window where HRT has the most protective effect on bone mineral density

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Although the study focused on a specific bacterial infection, it is fair to assume that other pathogens may elicit a similar response [51]