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A recent study suggests that among individuals with LDL cholesterol 190 mg/dl, gene sequencing identified a monogenic FH mutation in only 100 mg/dL (455)

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Like wild-type CaMKK2, autophosphorylation of the T85S variant is also triggered by Ca 2+ -calmodulin binding, but fails to keep CaMKK2 in an activated state and instead causes a temporary loss-of-activity [33]