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FATP5 promotor polymorphism (rs56225452), representing a putative gain-of-function mutation in the FATP5 promotor, correlated with BMI-dependent hepatic steatosis in males with biopsy proven NAFLD ( n = 103) [20], suggesting that genetic variation may underlie part of the contribution of FATP5 in NAFLD
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pylori as a major actor in gastric carcinogenesis, although the position of other microbes present in the stomach requires further evaluation ( H
(Much of her approach is informed by MyLymeData, a patient-powered Lyme-disease research project.) Because no two chronic-Lyme patients are the same, treatments must be highly individualized
