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2.1.2 Molecular and cellular mechanisms underlying the pathogenesis of AD Amyloid plaques arise from the aggregation of A peptides, particularly A4 2 , which is harmful due to its tendency to form plaques and resist dissolving (Jarrett et al., 1993)

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In both studies, total cagrilintide exposure (AUC 0 ), C max and other pharmacokinetic parameters were similar across groups with no consistent patterns observed with renal or hepatic impairment