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Boosts Immune System 3

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This imbalance leads to the infiltration of M1 macrophages, forming CD11c+ crown-like structures (CLS) around dead cells in WAT, resulting in the accumulation of high amounts of lipids within macrophages and promoting inflammation [99, 101]

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Under normal conditions in senescent cells, FOXO4 interacts with p53 and retains it in the nucleus, preventing p53 from translocating to mitochondria and executing its intrinsic pro-apoptotic programme