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This leads to the release and cytoplasmic translocation of p53, which can subsequently activate transcription-independent pro-apoptotic signaling (Figures 6A,B)

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This interaction can occur by itself, but occurs at a much faster rate with the assistance of a group of enzymes call the Glutathione-S-Transferases (GST)(figure1) (5)

The team, led by graduate fellow Nicole DelGaudio and postdoctoral fellow Hsi-wen Yeh, then analyzed the metabolites in these organelles to reveal how mitochondrial metabolites shift when cancer cells colonize new sites

doi: 10.2165/00128071-200405050-00003