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Common side effects (affecting up to 1 in 10 people) include abdominal pain, dyspepsia, and decreased appetite

Furthermore, such interactions extend beyond CYP3A4

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Recurrent associations between dysbiotic microbial patterns and poor metabolic response further support the hypothesis that gut microbiota may contribute to interindividual differences in response to incretin-based therapies, although definitive mechanistic evidence in humans remains limited (136), and causality between specific microbial taxa, functional pathways, and GLP-1 RA responsiveness cannot be inferred

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