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1994;304 ( Pt 2)(Pt 2):62533

Primary Cellular Pathways Nicotinic Acetylcholine Receptor Antagonism Neuromuscular Blockade The primary mechanism of Syn-AKE involves binding to the epsilon subunit of muscular nicotinic acetylcholine receptors (mnAChR), preventing acetylcholine from binding and initiating muscle contraction[1]

High-throughput profiling of off-target DNA cleavage reveals RNA-programmed Cas9 nuclease specificity

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Trial NCT01845493 consisted of a pilot study about the effect of SFN administration (in the form of BS homogenate) on Nrf2 and Phase II enzymes induction