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Although topical glucocorticoids effectively attenuate inflammatory dermatoses, they concurrently suppress the expression of lipid-synthesizing enzymes and antimicrobial peptides (AMPs), ultimately compromising epidermal permeability and innate immune barrier function [232, 233]

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This review further highlights the importance of considering PPAT-prostate interorgan communication as a critical component in prostate carcinogenesis, particularly given the key role that the PPAT can have in modulating the TME towards fuelling cancer development, which can be exacerbated by the influence of obesogenic EDCs Fig