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If similar mechanisms operate in retinal vessels, dual agonism might be associated with reduced transient hypoperfusion, believed to trigger early worsening

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As underlying mechanisms, it has been reported that GLP-1 causes renal vasodilatation, increases renal blood flow, increases glomerular filtration rate, inhibits the Na + /H + exchanger 3 in proximal tubule and decreases sodium, bicarbonate and water reabsorption [12, 14]

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