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The drug works by delaying gastric emptying and increasing feelings of fullness through hypothalamic pathways

This dual mechanism, eating less while burning more, accounts for the superior weight loss outcomes observed in clinical trials

Br J Haematol 2004

10.1186/s12943-020-01258-7 Summary Keywords ulcerative colitis, GHK-Cu, SIRT1/STAT3 pathway, co-culture system, network pharmacology, molecular docking Citation Mao S, Huang J, Li J, Sun F, Zhang Q, Cheng Q, Zeng W, Lei D, Wang S and Yao J (2025) Exploring the beneficial effects of GHK-Cu on an experimental model of colitis and the underlying mechanisms

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Avoid using the intravenous route