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These findings suggest that modulating ferroptosis pathways offers targeted benefits for the coronary microvasculature, addressing a specific component of coronary heart disease that often persists despite successful epicardial coronary intervention

Overexpression of EPAC2, on the other hand, dampened senescence and promoted the growth of transformed melanocytes (Fig

Non-responders had a significantly higher mean number of steps per day pretreatment compared to non-responders (4,246 2,888 vs

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