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There is a strong case that apoB is biologically closer to the driver of atherosclerosis than LDL-C [1], [2]

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The ability of AtGLRs to form diverse oligomeric assemblies likely contributes to their responsiveness to a broad spectrum of metabolite-derived ligands, including amino acids, organic acids, and secondary metabolites, thereby expanding their signalling flexibility

Kataranovski, M., Vlaki, M., Kataranovski, D., Toi, N., Mandi-Radi, S