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Mitochondrial dysfunction can activate the mitochondrial unfolded protein response (UPRmt) and integrated stress response (ISR), enhancing cellular stress adaptation and conferring resistance to chemotherapy and targeted therapies [41, 42]

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The activation was associated with significant IB degradation and nuclear translocation of NF-B, resulting in increased transcription of pro-inflammatory mediators (Metnitz et al., 1999)

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