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In contrast, no improvement in survival rate was observed in mice treated with H 2 S donor sodium hydrogen sulfide, indicating that the anti-inflammatory effects of NAC-S2 are far superior to those of H 2 S donor ( Figure 5 )

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Given the central role of citrate in the tricarboxylic acid cycle, its depletion may signal compromised aerobic respiration, possibly driven by heightened oxidative stress or inflammation, two processes well documented in epileptogenesis 7,12,13,14,15