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Mendelian randomisation in cardiovascular research: an introduction for clinicians

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Abnormal post-translational modifications of ion channels, due to impaired PI3K-Akt or MAPK signaling, further contribute to network hyperexcitability

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Further histopathological analyses showed that the elevation in liver damage biomarkers observed in the Gclc L-KO mice was not associated with increased markers of apoptosis but was associated with increased markers of necroptosis (Fig

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