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Salim Kanaan, [email protected] These authors have contributed equally to this work and share first authorship Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Limitations: Lower bioavailability and inconsistent dosing

Receptor-independent effects of melatonin in GC: The induction of intrinsic apoptosis (increased Bax, Bak, and Bax/Bcl-2 ratio), sensitization to extrinsic apoptosis (upregulation of TNFRSF10A and TNFRSF10B), activation of ER stress pathways (IRE1, spliced Xbp1, phosphorylated PERK, and CHOP), modulation of autophagy markers (increased LC3B and Beclin1, decreased p62), and induction of necroptosis (RIPK1 and MLKL upregulation) are closely associated with intracellular stress responses

Keine Nahrungsergnzung ersetzt jedoch die potenzielle Wirkung von Thymosin-Analoga

cycle-based Human evidence level Moderate Clinical trials