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Bantle, C

In addition, we also collected neutrophils gene datasets with other autoinflammatory and autoimmune diseases, including systemic lupus erythematosus (SLE, GSE153781, unpublished data), adult-onset Stills disease (AOSD, GSE150996) [13], and systemic juvenile idiopathic arthritis (SJIA, GSE122552) [14] to compare the oxidative burst of neutrophils

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PLoS ONE 7 , e50005 (2012)

Our prior pharmacokinetic studies revealed rapid metabolism of BPC157 into small peptide fragments, ultimately yielding single amino acid residues (predominantly proline) in plasma, urine, and feces [11]