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[1] [2] Short-chain fatty acids (primarily acetic, propionic, and butyric acids), which microganisms form in the intestines, bind to the FFAR2 and FFAR3 receptors on K cells and L cells to stimulate their respective production and secretion of GIP [3] and GLP-1
The available studies differ in drug type, dose, indication, timing of exposure, comparator structure, and baseline maternal metabolic risk, and the number of exposed pregnancies remains insufficient to exclude uncommon but clinically important harms (132, 133)
Researchers following this literature should reference the published Phase 2 data as the current evidence base while monitoring Phase 3 publications for updated dosing information
