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Inhibition of AKT induces p53/SIRT6/PARP1-dependent parthanatos to suppress tumor growth

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High and unstable blood glucose levels will promote tumor cell growth [48], so a reduction in glucose following L-carnitine treatment might have been beneficial to inhibit tumor growth in the CT26 tumor-bearing mice

High glucose availability limits the ability of fatty acids to get inside the mitochondria, so fatty acid oxidation goes down and the mitochondria predominantly oxidize glucose for energy