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Peripheral and central effects of circulating catecholamines
Antibody blockade of activin type II receptors preserves skeletal muscle mass and enhances fat loss during GLP-1 receptor agonism
This supports a multifactorial model in which semaglutide may amplify ischemic susceptibility in predisposed individuals, such as those with small BMO, ODD or otherwise crowded optic nerve head, rather than acting as a direct neurotoxic agent
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Semaglutide may help counteract this by supporting the growth and survival of these cells
