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Phosphoproteomic analysis of neuronal cell death by glutamate-induced oxidative stress

reported that UC and CD patients with active disease have elevated levels of bioactive GLP-2 compared with healthy subjects (Xiao et al

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These and other modifications presumably prevent an efficient interaction with GSH as reflected by the absent GSH-dependent reduction of oxidized roGFP2 in the presence of AtGrxS15

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Epilepsy in Leigh syndrome with mitochondrial DNA mutations