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Finally, a third group reflected SGLT2i influences on metabolites, comprising reversal of abnormalities in long-chain fatty acid uptake, provision of ketonaemia as an alternative fuel that augments autophagic flux, improvement in iron homeostasis (increasing the bioreactive Fe pool while lowering ferritin and hepcidin), and mitigation of cytosolic accumulation of harmful glucose and lipid by-products, accompanied by an overall enhancement of mitochondrial oxidative metabolism

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2 Within the gastrointestinal (GI) tract, activation of GLP-1R reduces gastric muscle contractions and slows gastric emptying, prolonging satiety and reducing post-prandial glucose spikes

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[3] [4] El exceso de acetil-CoA provoca que ms hidratos de carbono sean usados para obtener energa a expensas de los cidos grasos