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Neurons use mitochondria to produce adenosine triphosphate (ATP) to power the ion pumps which are concentrated in the pre-synaptic terminals and required to restore the membrane potentials after depolarization and neurotransmitter release (Mattson & Liu, 2002)

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In-depth research on the relationship between UA and Akt/GSK3 signaling pathway provides a promising avenue for therapeutic interventions aimed at mitigating the progression of neurodegenerative conditions

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