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Specifically, AMPK can inactivate ACC by phosphorylation, resulting in reduced activity of malonate monoacyl coenzyme A (CoA) and reducing allosteric inhibition of CPT1, thereby enhancing the activity of CPT1, which transports long-chain fatty acids from carnitine to mitochondria for -oxidation [17]

42 LessingF.KniemeyerO.WozniokI.LoefflerJ.KurzaiO.HaertlA.et al (2007)

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9) (Narayanasamy and Murugesan 2014

That's because cancer cells, like stem cells, rely on telomerase to maintain a ceaseless rate of replication