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Schizophrenia bulletin 41:10161020

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After 24 hours of postovulatory aging: Intracellular ROS levels decrease Mitochondrial membrane potential remains stable mtDNA copy number stays consistent Cortical granule abnormalities and spindle defects are reduced DNA damage (H2AX) and early apoptosis markers decline Early apoptosis decreases Fragmentation rates fall during parthenogenetic activation These results highlight Epithalons role in combating oxidative stress and mitochondrial decline in reproductive cells

p53-pS15 combined with FOXO4 in the nucleus of senescent cells inhibits p53 into the cytoplasm to mediate apoptosis and halt the cell cycle 9,11