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The integration of all three pathways is proposed to create a complementary pharmacological profile: appetite suppression and improved insulin secretion from GLP-1 and GIP agonism, combined with enhanced energy expenditure and hepatic lipid metabolism from glucagon receptor activation

You should not feel too sick to eat, work, exercise, or function normally

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Available from: Balaban DH, McNamara S, Wallman K, et al

Stable labeled microspheres to measure perfusion: validation of a neutron activation assay technique

All of those 3 things are done by GLP-1 and this is a version of GLP-1