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1C), strongly suggesting that Prk1 is a novel regulator of metal sensitivity

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The fragment pools produced (libraries) were diluted to the concentration of 4 nM, denatured, and diluted to 5 pM

The mechanism may be related to inhibiting ATG16L1-mediated autophagy of normal colon epithelial cells, promoting ROS accumulation and NLRP3 inflammasome assembly

Methods Study design An 8-week randomized, double-blind, placebo-controlled parallel study was conducted at a single center, KGK Synergize Inc., in London, ON, Canada, between January 12, 2015 and June 19, 2015

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