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Succinyladenosine accumulation has been reported in inborn errors of metabolism that disrupt purine metabolism and mitochondrial function, such as adenylosuccinate lyase (ADSL) deficiency and fumarase deficiency [49, 50], and its consistent upregulation in MMA may reflect secondary effects on nucleotide metabolism or mitochondrial energy balance, representing an underexplored aspect of MMA pathophysiology

PGC1/CEBPB/CPT1A axis promotes radiation resistance of nasopharyngeal carcinoma through activating fatty acid oxidation

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The clinical incidence is low

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