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Oxidative stress and damage occurs when antioxidant defense mechanisms fail to effectively counter endogenous or exogenous sources of reactive oxygen species

doi: 10.1021/acs.jmedchem.5b00726 96 DeaconCFKnudsenLBMadsenKWibergFCJacobsenOHolstJJ

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1, the post-infarction state is marked by a dysbiotic shift characterized by the depletion of beneficial SCFAs and the accumulation of pro-inflammatory mediators like TMAO and LPS, thereby exacerbating gut inflammation and potentially accelerating the pathological progression of cardiac injury 20,21

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A cross-sectional study that compared 45 cases of NAFLD with 30 healthy controls found lower vitamin B12 in patients with grade 2 and grade 3 of liver steatosis 8