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Interventions: Cagrilintide, semaglutide, placebo Primary Outcome Measures: Relative change in body weight

219 In NUT midline carcinoma, aberrant BRD4 chromatin retention disrupts autophagy and lysosomal function, revealing therapeutic vulnerability

Binding to GHSR-1a stimulates GH release via a calcium-dependent, cAMP-independent intracellular pathway, distinct from GHRH-mediated signalling

VacA generates a protective intracellular reservoir for Helicobacter pylori that is eliminated by activation of the lysosomal calcium channel TRPML1

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