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We propose that mitohormesis may be optimally activated by strategies that facilitate a balanced oscillation between challenging (but not excessive) stressor phases, which temporarily disrupt mitochondrial biology, and complete (but not excessive) recovery phases, which provide adequate time for mitochondria to repair and recalibrate prior to the next stressor (Table 1)

Das Gupta K, Shakespear MR, Iyer A, Fairlie DP, Sweet MJ

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It is not hard to extrapolate, then, that patients with multiple contributing factors for reduced glutathione levels may experience more severe disability and worse outcomes due to the loss of the antioxidant