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Endogenous GLP-1 mediates postprandial reductions in activation in central reward and satiety areas in patients with type 2 diabetes

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In turn, in PNA mice, treatment with GLP1/E caused a significant reduction in CPOX and BCAP31 expression, and increased levels of CAMK2B (Fig

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The slowed gastric emptying combined with altered gut motility creates unpredictable effects on colonic transit

Characterising the burden of chronic kidney disease among people with type 2 diabetes in England: a cohort study using the clinical practice research datalink